From Scientific American, The Scientific Secret of Fluffy Pancakes.
I really like this bit of using the scientific method to investigate why it’s always recommended to not mix the pancake better too much. It’s a great way to learn about foods, ingredients, and why we cook things in certain ways.
Where can I find more ideas like this to try? (Preferably with allergy-friendly foods).
Andrew Jacobs, for the Mew York Times – In Sweeping War on Obesity, Chile Slays Tony the Tiger
Nutrition experts say the measures are the world’s most ambitious attempt to remake a country’s food culture, and could be a model for how to turn the tide on a global obesity epidemic that researchers say contributes to four million premature deaths a year.
The required changes to the food labelling and logos are quite profound.
Bee Wilson – First Bite: How We Learn to Eat
I’m only half way through the book, but it’s very thought-provoking and relevant to us right now, since our toddler is just nearing the age of 3. Chapter 4 on Feeding discusses several styles of feeding children that researchers have divided them into:
- Uninvolved: low warmth and low demands
- Authoritarian: low warmth and high demands
- Indulgent: high warmth and low demands
- Authoritative: high warmth and high demands
Where ‘warmth’ is described as being the level of sensitivity to the child’s needs. I think of myself as being somewhere between Indulgent and Authoritative. I tend to indulge our son in many of his wants but he tends to be quite good in his ability to accept small indulgences (one or two mini chocolate chips, for example) as sufficient, so it’s never a worry for me. At the dinner table; however, I tend to ask him several times if he wants to try a food, or if he wants to finish his soup or if he’ll have one more bite of something. Especially after he starts getting down from his chair to go play. The danger being that the high demands may skew his liking of certain foods that we serve. Regardless, the take-away is best stated by Wilson:
The art of feeding, it turns out, is not about pushing “one more bite” into someone’s mouth, however healthy the food. Nor is it about authoritarian demands to abstain from all treats. It’s about creating a mealtime environment where – as in Clara Davis’s feeding experiment – those who are eating are free to develop their own tastes, because all of the choices on the table are real, whole food.
Aaron E. Carroll for the New York Times, The Upshot – Simple rules for healthy eating:
All of these rules are subtly trying to get you to be more conscious of what you’re eating. It’s far too easy these days to consume more than you think you are, or more than you really need, especially when eating out. I’ve found that it’s impossible to tell any one person how much they should be eating. People have varying requirements, and it’s important for all of them to listen to their bodies to know when they should eat, and when they should stop.
I’ve found that making change is hard. How many times have I started running, only to stop a few weeks later due to illness and not getting back into it when I’m better? But we have made the change in our diets. Slowly and over time (in the last 4-5 years) we’ve made small changes that have resulted in weight loss (30+ lbs for me over that time), reduction in cholesterol levels and overall feeling better about my body image.
Julia Belloz at Vox – We’ve long blamed carbs for making us fat. What if that’s wrong?
The main scientific model behind the low-carb approach is the “carbohydrate-insulin hypothesis,” which journalist Gary Taubes, Harvard professor David Ludwig, and others have extensively promoted. It suggests that a diet heavy in carbohydrates (especially refined grains and sugars) leads to weight gain because of a specific mechanism: Carbs drive up insulin in the body, causing the body to hold on to fat and suppress calorie burn.
But what’s often lost in all the boosterism around the low-carb approach is that it is still an unproven hypothesis in science.
So they did a study to show whether a low-carb diet can actually help to reduce weight. 17 overweight or obese men in a metabolic chamber for 4 weeks and fed either a high carbohydrate diet or ketogenic diet with the same number of calories. Results? No increased body fat loss with the ketogenic diet.
But as Bazinet points out, “The study … doesn’t see any [relationship between a decrease in insulin and an increase in fat loss]. Show me a better study that supports this.”
There isn’t any, he added.
Tobias urged dieters not to lose sight of the bigger picture. “Low-carb versus low-fat should not be the focus for people selecting a weight loss diet.” The focus, she said, should be on improving the quality of food that people eat instead.
Jill Eisen for the CBC – Fat and Sugar, Part 1
First, fat was the dietary bad guy. We were warned back in the 1980s to cut back on eggs, meat and full-fat dairy to avoid heart disease. So we started eating more bread, rice and pasta and fat-free snacks. But we got sicker and fatter. Now sugar is the bad guy. Contributor Jill Eisen explores the complex, and sometimes contradictory, science of nutrition — and tries to find clarity amidst the thicket of studies and ambiguous research.
There’s also a great list of research references and a reading list at the link, for more information.
Part 2 airs on June 22, 2016.
The National Academy of Sciences has published a report – Genetically Engineered Crops: Experiences and Prospects:
Human Health Effects:
GE crops and foods derived from them are tested in three ways: animal testing, compositional analysis, and aller-genicity testing and prediction. Although the design and analysis of many animal-feeding studies were not optimal, the many available animal experimental studies taken together provided reasonable evidence that animals were not harmed by eating foods derived from GE crops. Data on the nutrient and chemical composition of a GE plant compared to a similar non-GE variety of the crop some-times show statistically signiﬁcant differences in nutrient and chemical composition, but the differences have been considered to fall within the range of naturally occurring variation found in currently available non-GE crops.
Many people are concerned that GE food consumption may lead to higher incidence of speciﬁc health problems including cancer, obesity, gastrointestinal tract illnesses, kidney disease, and disorders such as autism spectrum and allergies. In the absence of long-term, case-controlled studies to examine some hypotheses, the committee examined epidemiological datasets over time from the United States and Canada, where GE food has been consumed since the late 1990s, and similar datasets from the United Kingdom and western Europe, where GE food is not widely consumed. No pattern of differences was found among countries in speciﬁc health problems after the introduction of GE foods in the 1990s.
Anna Maria Barry-Jester – Is Gut Science Biased?:
The vast majority of data on microbiomes across science is still mostly coming from people in the U.S., Canada and Europe, and having too narrow a sample of the population means we could be looking at the proverbial trees when what we need to understand is the whole forest.
Much of what we know about gut microbiomes is biased towards what can be easily measured – the guts of people in the western world. I’d take articles touting the benefits of this or that diet on gut microbiome with a grain of salt.
Derek Lowe – Glyphosate and Cancer:
I went into one of those in detail here, and after looking into the case that it makes, I am willing to dismiss out of hand anything else Seneff has to say on the subject. It’s that bad. You will hear that “MIT researchers” have “proven” that glyphosate does X and Y and Z, and that this work is “published in peer-reviewed journals”, but nothing like that is true. Seneff has done no actual studies on glyphosate; she doesn’t work in a lab. Those papers are rehashes of stuff from the literature, piles of speculation and dot-connecting, and they’re invariably published in low-quality pay-to-play journals that do little or no actual refereeing of their contents. And their content is yet another problem – as shown in that link above, the paper that goes on and on about glyphosate’s effect on gut bacteria does not manage to cite any of the papers that have studied. . .glyphosate effects on bacteria. It not only doesn’t cite them, it seems to pretend that this research does not even exist, probably because all these papers contradict the fundamental ideas that Seneff’s tower of speculation is built on. She’s going around now saying that half of all children are going to be autistic (because of glyphosate), and that it’s also a root cause of not only cancer, but Alzheimer’s and a whole list of other diseases. If your knowledge of glyphosate’s toxicology comes only from reading the Seneff papers, I feel pity for you, because you have a lot of work ahead of you if you want to actually understand anything about it.
In considering the risk of cancer due to glyphosate, he compares it to the recent articles on bacon/cured meats and risks of cancer:
…that’s basically what happened recently with the IARC and its announcement on bacon being a cause of cancer. Under real-world conditions, eating a normal amount of bacon raise your risk of colorectal cancer by an amount too small to consider. But it does appear to be raising it by a reproducible, measurable amount, and therefore bacon (and other processed meats) are in the IARC’s category 1
And the conclusion:
So that’s the state of the art: there is, from what I can see, nothing very clearly linking glyphosate to human cancer. There’s certainly room for more evidence to come in, though, and it looks like we’re going to need it, because this is a topic that’s never going to go away until we have more data.
Elina L. Niño – Deciphering the mysterious decline of honey bees:
Scientists now agree that CCD was likely caused by a combination of environmental and biological factors, but nothing specific has been confirmed or proven. CCD is no longer causing large-scale colony death in North America, but beekeepers all over the United States are still reporting troubling colony losses – as high as 45 percent annually.
I repeat, nothing specific has been confirmed or proven.
Many people who are not beekeepers or growers want to know how they can help. One easy step is to grow forage plants, especially varieties that bloom at different times during the year. For suggestions, see our Häagen-Dazs Honey Bee Haven Plant List.
Second, reduce your pesticide use for gardening and landscaping, and follow guidelines to reduce bee exposure. Finally, you can support local beekeepers by buying their honey.
Ultimately, however, making our society more pollinator-friendly will likely require some drastic and long-term changes in our environmental and agricultural practices.
Good points. We definitely need to plant more forage plants in our yard.